Klopefir Mix

v Effective against most weeds in cereal crops, including the most harmful ones, such as Euphorbia vinifera, Galium tenacious, genus Ichthyos, Cirsium spp., and Sow Thistle.

v Due to the reduced consumption rates of the components, the preparation does not have a negative impact on the treated crops and can be used in a wide range of  - from the two-leaf stage until the first internode appears.

v Due to the rapid decomposition of tribenuron-methyl in the soil, there is no risk of aftereffects, so it can be used without restrictions in all types of crop rotations.

Mechanism of Action
Esters of 2,4-D acid and clopyralid are synthetic forms of plant growth hormones. Once in the plant, they are quickly redistributed (throughout the xylem and phloem) and replace natural plant hormones, thereby blocking their functions. In this case, oversaturation with synthetic hormones occurs, which leads to disruptions in the process of photosynthesis and cell division in the meristem, causing abnormal deformation of leaves, significant disruption of weed growth and their subsequent death.
Tribenuron-methyl penetrates through the leaves and partially through the root system into the plant, blocking the synthesis of the enzyme acetolactate synthase, which is involved in the synthesis of essential amino acids valine, leucine and isoleucine. Suppression of this enzyme blocks cell division, leading to rapid cessation of weed growth, yellowing, and then death.

Symptoms of exposure
The main symptoms of exposure are: uneven growth of above-ground organs, various types of deformation, leaf discoloration, shortening of internodes, reddening of veins, leaf chlorosis, death of growth points, tissue necrosis.

Speed ​​of action
The components of KLOPEFIR MIX enter the plant through the leaves within 2-3 hours after treatment, actively moving within it, concentrating in areas of active cell division (growth points). The growth of sensitive weeds and competition with the crop are inhibited within a few hours after treatment, and cease within 24 hours.
Susceptible weeds usually die within 3-7 days, with complete death occurring within 2-3 weeks. Under unfavorable weather conditions, herbicide activity may be reduced.

Spectrum of action
Susceptible weeds: hemlock, common ragweed, radiant biphora, field thistle, spotted hemlock, common wart-bearing grass, blue cornflower, veronica, field vetch, field sparrow-foot, crow's foot, geranium (species), knotweed (species), field mustard, tartary buckwheat, rhododendron (species), distochnik (species), medicinal sweet clover, common datura, medium chickweed, clover (species), common bugleweed, commelina, weed hemp, nettle (species), common ragwort, common cockle, kuray, spreading orach, sticky grass (species), burdock (species), common toadflax, field buttercup, alfalfa (species), pigweed (species), Canadian fleabane, Tatar milkweed, vine spurge, field forget-me-not, shepherd's purse, pygmy sedge (species), cleavers, weed sunflower, wormwood, Rapeseed, wild radish, scentless chamomile, common winter cress, wild pansy, vetch (species), retroflexed amaranth, field pennycress, deadnettle species.
Moderately sensitive: field bindweed, galinsoga (species), hibiscus trifolatum.

Protective action period
Does not have a soil effect, it affects only the weeds that were present at the time of treatment. If the recommendations are followed, one treatment is sufficient to effectively suppress dicotyledonous weeds until the end of the growing season, at